(FIGURE 1) White spots are seen at the level of the deep retina. Fluorescein angiography (FA) shows pinpoint areas of hyperfluorescence in a circular or wreath distribution, which is characteristic of MEWDS.
(All images courtesy of Glenn J. Jaffe, MD, unless otherwise noted)
(FIGURE 2) Fundus photograph (A) and FA (B) of patient with birdshot retinochoroidopathy. The FA is somewhat unremarkable and may have a washed out or featureless appearance. The spots seen on color fundus photography are not seen clearly.
(FIGURE 3) Fundus photograph (A) and FA (B) of patient with punctate inner choroidopathy. Hyperfluorescence corresponds to the spots seen on color fundus photography.
(FIGURE 4) Fundus photograph (A) and FA (B, C) of a patient with
multifocal toxoplasmosis. Lesions were hypofluorescent early, and
stained late.
(FIGURE 5) FA of a patient with sarcoidosis. The hyperfluorescence corresponds to the neovascularization. There is adjacent hypofluorescence corresponding the vascular occlusion.
(FIGURE 6) ICG angiography in a patient with multifocal choroiditis and panuveitis (A) and MEWDS (B).
(FIGURE 7) OCT angiography shows blood fl ow by obtaining sequential images at identical locations. (Courtesy of Kester Nahen)
(FIGURE 8) Example of Type-2 Cnv imaged by FA, ICG, structural OCT, and OCT-A. With OCT-A, the depth of the Cnv lesion can be determined. (Images courtesy of Kester Nahen and Giovanni Staurenghi)
(FIGURE 9) Multi-modal example of a patient with serpiginous choroiditis. The fundus photograph (A) and the FA (B) show hypofl uorescent lesions. The OCT (C, D) shows the signal penetration because of the atrophic lesions. Fundus autofl uorescent imaging (E) is very helpful, as it indicates hyperautofl uorescent areas (arrows) which correspond to active lesions that require further treatment.